Tricuspid atresia is a congenital cyanotic heart disease. The main pathological change is tricuspid atresia, tricuspid missing, patent foramen ovale, atrial septal defect, mitral valve and left ventricular hypertrophy, or poor development of the right ventricle. Patients have reduced amount of blood for the pulmonary circulation. Most cases have cyanosis, shortness of breath, rapid breathing, frequent lung infection and congestive heart failure from birth. In addition, there may be signs of enlarged liver, edema, enlarged neck veins and pulmonary edema. Most children with tricuspid atresia can live to adulthood after surgery. However, they often need follow-up surgeries.
How is Tricuspid atresia treated in practice?